GLP-1 and the regulation of carbohydrate intake in the rat
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GLP-1 and the regulation of carbohydrate intake in the rat

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Published by National Library of Canada in Ottawa .
Written in English


Book details:

Edition Notes

Thesis (M.Sc.) -- University of Toronto, 2000.

SeriesCanadian theses = -- Thèses canadiennes
The Physical Object
FormatMicroform
Pagination2 microfiches : negative. --
ID Numbers
Open LibraryOL20799305M
ISBN 100612541584
OCLC/WorldCa50755911

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A role for glucagon-like peptide-1 in the central regulation of feeding Article (PDF Available) in Nature () February with Reads How we measure 'reads'. Glucagon-like peptide (GLP)-1 is an incretin hormone, released from the gut into the blood stream in response to intestinal nutrients. Endogenous GLP-1 levels increase following food intake, particular of carbohydrate (15, 16). These studies suggest a role for GLP-1 in mediating the effects of carbohydrate (specifically glucose) on by: Glucagon-Like Peptide 1 (GLP-1) GLP-1 physiology. GLP-1 is secreted primarily from entero-endocrine L cells located in the distal jejunum and ileum, and its release is stimulated by carbohydrate and fat intake (Madsbad, ). Secretion of GLP-1 in response to food intake occurs rapidly after food ingestion (within 10–15 min) with a second.   Interestingly, suppression of the pre-meal GLP-1 rise leads to lower, rather than greater food intake. This is inconsistent with GLP-1’s reported anorexic effect, but may reflect the lack of suppression of orexigenic signals (65, ). Second, glucose can pass through SGLT1 on L-cells depolarizing the cell and releasing by:

GLP-1 acts to inhibit food intake. Acute GLP-1 injection into the third or fourth ventricles or into the PVN reduces calorie intake, and chronic central administration decreases weight gain in rodents. Peripheral injection also reduces food intake and activates c-fos in the brain stem (, ). Thus peripheral GLP-1 may influence energy Cited by: This review aimed to describe the potential mechanisms by which incretin hormones could mediate the relationship between glycemic index and cardiometabolic diseases. A body of evidence from many studies suggests that low glycemic index (GI) diets reduces the risk for type 2 diabetes and coronary heart disease. In fact, despite the extensive literature on this topic, Author: Teresa Salvatore, Riccardo Nevola, Pia Clara Pafundi, Lucio Monaco, Carmen Ricozzi, Simona Imbriani.   Goals / Objectives The goal of the proposal is to design carbohydrate-based functional foods for energy balance and optimum health through elucidation of physiological responses to dietary carbohydrate, particularly slowly digestible glycemic carbohydrates and their interaction with fermentable dietary fibers. Thus, it responds directly to research needs . Take a look at the scientific evidence-based literature to support the statements we make in the book! Mastering Diabetes Book Scientific References. The Revolutionary Method to Reverse Insulin Resistance Permanently in Type 1, Type , .

  M. Navarro, et al., Changes in Food Intake Induced by GLP-1() amide in the Rat, Abstracts of the 15th International Diabetes Federation Congress, Nov. , Kobe, poster presentation 11A5PP M.   Pancreatic regulation of glucose homeostasis. (GLP)-1 and glucose-dependent including the regulation of carbohydrate and fatty acid metabolism and may be considered as a myokine due Cited by: This study determined the effects of increasing loads of intraduodenal (ID) dairy protein on plasma amino acid (AA) concentrations, and their relationships with serum insulin, plasma glucagon-like peptide-1 (GLP-1) and energy intake. Sixteen healthy men had concentrations of AAs, GLP-1 and insulin measured in response to min ID infusions of hydrolysed whey protein administered, Cited by:   Turton et al. () specifically state that the effects of GLP-1 on body weight and food intake are caused only by administration of GLP-1 directly in the cerebroventriculum, that intraperitoneal administration of GLP-1, even at relatively high does, does not affect early dark-phase feeding, and that GLP-1 fragments are inactive when.